Hyperbaric Oxygen Therapy Support Stroke Recovery and Stroke Prevention
Stroke is the fourth leading cause of death in the U.S. and has remained a leading cause of adult disability with 800,000 cases reported every year.
Hyperbaric oxygen therapy (HBOT) has been shown to help prevent and treat stroke through a number of different mechanisms.
HBOT can be used to precondition the brain by stimulating angiogenesis, thus producing more circulatory pathways throughout the brain and potentially decreasing the chance of stroke. Also, this has been shown to benefit post-stroke patients, as compromised blood flow to the affected brain tissue (penumbra) and its functionality is recoverable by introducing increased levels of oxygen.
Benefits of Hyperbaric Oxygen Therapy for Patients Recovering from Stroke and for Stroke Prevention
Studies have demonstrated the following therapeutic benefits of hyperbaric oxygen therapy for patients recovering from stroke. HBOT may also help prevent stroke.
Supporting Stroke Recovery using Hyperbaric Oxygen Therapy
- Faster Overall Recovery
- Improves Vision and Speech
- Reduces Paralysis
- Accelerates Gross/Fine Motor Skills
- Increases Penumbra Tissue Recovery
- Stimulates Angiogenesis for Faster Reclamation
- Promotes Neurogenesis
- Enhances Stem Cell Proliferation and Mobilization
- Escalates Neuroplasticity
- Alleviates Spasticity
How Hyperbaric Oxygen Therapy May Prevent Strokes
- Stimulates Angiogenesis
- Ameliorates Atherosclerosis
- Decreases Oxidative Stress in the Brain
- Preconditions the Brain to Enhance Neuroprotective Properties
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The following research article highlights how hyperbaric oxygen therapy supported the recovery of post-stroke patients.
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Hyperbaric Oxygen Therapy Supports Post-Stroke Patients
In January 2013 a prospective, randomized, controlled trial focused on the introduction of hyperbaric oxygen therapy (HBOT) to post-stroke patients. A total of 59 participants, who had suffered a stroke 6 to 36 months prior to inclusion and had at least one motor dysfunction, were randomly assigned to treated and cross-over groups. The treated group received two months of 40, one-hour HBOT sessions. Whereas the cross-over group was evaluated after one month with no HBOT and again after one month following HBOT, utilizing the same treatment protocol. The evaluating physicians found that neurological function, brain activity and quality of life of all treated patients improved after HBOT. Results of SPECT imaging directly correlated with clinical improvements and indicated that HBOT can lead to significant neurological improvements in post-stroke patients, even at chronic late stages. The observed clinical improvements indicated that neuroplasticity can be activated long after damage onset.